3-AD® Prohormone

If 3-AD is a prohormone, explain why some users claimed their blood work showed an increase in testosterone levels. 

1. Indirect Upregulation of Testosterone Production

3-AD (Dehydroandrosterol) does not directly convert into testosterone, but it may still influence androgenic activity in ways that stimulate natural testosterone production:

• Reduced Cortisol → Increased Testosterone

• 3-AD is known to have anti-catabolic properties, meaning it may reduce cortisol levels.

• Lower cortisol levels allow for higher testosterone production, as cortisol and testosterone have an inverse relationship.

• Increased Androgen Receptor Sensitivity

• If 3-AD binds to androgen receptors, it may cause the body to upregulate testosterone production to compensate.

• This is a common adaptive response seen with milder androgenic compounds.

• Inhibition of Aromatase or SHBG Modulation

• If 3-AD affects aromatase or Sex Hormone Binding Globulin (SHBG) levels, it could lead to higher free testosterone levels without increasing total testosterone directly.

• Lower SHBG = More bioavailable testosterone in the bloodstream.

2. Potential Influence on Precursor Hormones

Since 3-AD is structurally related to DHEA, it is possible that it enhances precursor activity that contributes to testosterone levels:

• 3-AD May Influence Other Steroidogenic Pathways

• DHEA naturally contributes to testosterone production via metabolic intermediates.

• While 3-AD does not convert to testosterone, it may support the body’s natural DHEA pathways, leading to an indirect increase in testosterone.

• Upregulation of Steroidogenesis

• If 3-AD stimulates androgenic activity, the body may increase testosterone production in response to maintain hormonal balance.

3. Individual Physiological Response

• Some users may have naturally suppressed testosterone production before using 3-AD, and the supplement may have acted as a mild hormonal regulator.

• Genetics play a role—some individuals may be more sensitive to testosterone upregulation from androgen receptor activity.

Final Verdict: 3-AD’s Testosterone Boost is Indirect, Not a Conversion

• 3-AD itself does NOT convert into testosterone.

• The increase in testosterone levels could be due to:

✅ Reduced cortisol leading to better testosterone retention

✅ Increased androgen receptor sensitivity stimulating testosterone production

✅ Modulation of SHBG or aromatase activity affecting free testosterone levels

✅ A mild upregulation of steroidogenesis via DHEA-related pathways

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